Co-Authors:
Svoboda, J.A., Insect Physiology Laboratory, Agricultural Research Service, U.S. Department of Agriculture, Beltsville, 20705, MD, United States
Thompson, M.J., Insect Physiology Laboratory, Agricultural Research Service, U.S. Department of Agriculture, Beltsville, 20705, MD, United States
Herbert Jr., E.W., Bioenvironmental Bee Laboratory, Agricultural Research Service, U.S. Department of Agriculture, Beltsville, 20705, MD, United States
Shortino, T.J., Insect Physiology Laboratory, Agricultural Research Service, U.S. Department of Agriculture, Beltsville, 20705, MD, United States
Szczepanik-Vanleeuwen, P.A., Division of Radiological and Environmental Research, Argonne National Laboratory, Argonne, 60440, IL, United States
Abstract:
The honey bee, Apis mellifera, does not convert C28 and C29 phytosterols to cholesterol as found in most previous studies of phytophagous or omnivorous insects, but instead the workers and queens selectively transfer 24-methylenecholesterol, sitosterol and isofucosterol from their endogenous sterol pools to the brood larvae regardless of the sterol in the worker diet. Administering radiolabeled sterols by feeding and injection has made it possible to trace this selective transfer through a second generation of the honey bee. In further comparative sterol metabolism studies, the yellow fever mosquito, Aedes aegypti, was shown to be capable of dealkylating and converting a radiolabeled C29 dietary sterol ([14C] sitosterol) to cholesterol. Metabolic studies with several radiolabeled dietary sterols and an inhibitor of steroid metabolism in the yellow fever mosquito further verified this capability. © 1982 American Oil Chemists' Society.
תפריט נגישות
ניגודיות עדינה
מונוכרום
הדגשת קישורים
חסימת אנימציה
פונט קריא
סגור
איפוס הגדרות נגישות
להורדת מודול נגישות חינם
