נגישות
menu      
חיפוש מתקדם
תחביר
חפש...
הספר "אוצר וולקני"
אודות
תנאי שימוש
ניהול
קהילה:
אסיף מאגר המחקר החקלאי
פותח על ידי קלירמאש פתרונות בע"מ -
Lipid oxidation products and chick nutritional encephalopathy
Year:
1979
Source of publication :
Lipids
Authors :
ברטוב, עדו
;
.
Volume :
14
Co-Authors:
Budowski, P., Faculty of Agriculture, The Hebrew University of Jerusalem, Rehovot, Israel
Bartov, I., Division of Poultry Science, Agricultural Research Organization, The Volcani Center, Renovot, Israel
Dror, Y., Faculty of Agriculture, The Hebrew University of Jerusalem, Rehovot, Israel
Frankel, E.N., Northern Regional Research Center, Agricultural Research, Science and Education Administration, U.S. Department of Agriculture, Peoria, 61604, Illinois, United States
Facilitators :
From page:
768
To page:
772
(
Total pages:
5
)
Abstract:
Safflower oil and its distilled methyl esters were thermally oxidized and fed to young chicks in a vitamin E deficient diet. At a dietary level of 10%, the oxidized lipids caused more severe nutritional encephalopathy (NE) than the unoxidized methyl esters, indicating that factors other than dietary linoleic acid and vitamin E affect the development of NE. A polar lipid extract from oxidized methyl esters accelerated the induction of NE, as did the synthetic methyl esters of keto-octadecenoic and keto-octadecadienoic acids. Dicumarol exerted a protective action against NE. The possibility is discussed that conjugated keto-polyenoic fatty acids, provided by oxidized oils or formed endogenously in vitamin E deficiency, may play a role in causing NE. © 1979 American Oil Chemists' Society.
Note:
Related Files :
aging
Animal
animal experiment
Chickens
Dietary Fats
encephalomalacia
lipid
Lipids
Male
Oils
עוד תגיות
תוכן קשור
More details
DOI :
10.1007/BF02533514
Article number:
Affiliations:
Database:
סקופוס
Publication Type:
מאמר
;
.
Language:
אנגלית
Editors' remarks:
ID:
30959
Last updated date:
02/03/2022 17:27
Creation date:
17/04/2018 00:58
Scientific Publication
Lipid oxidation products and chick nutritional encephalopathy
14
Budowski, P., Faculty of Agriculture, The Hebrew University of Jerusalem, Rehovot, Israel
Bartov, I., Division of Poultry Science, Agricultural Research Organization, The Volcani Center, Renovot, Israel
Dror, Y., Faculty of Agriculture, The Hebrew University of Jerusalem, Rehovot, Israel
Frankel, E.N., Northern Regional Research Center, Agricultural Research, Science and Education Administration, U.S. Department of Agriculture, Peoria, 61604, Illinois, United States
Lipid oxidation products and chick nutritional encephalopathy
Safflower oil and its distilled methyl esters were thermally oxidized and fed to young chicks in a vitamin E deficient diet. At a dietary level of 10%, the oxidized lipids caused more severe nutritional encephalopathy (NE) than the unoxidized methyl esters, indicating that factors other than dietary linoleic acid and vitamin E affect the development of NE. A polar lipid extract from oxidized methyl esters accelerated the induction of NE, as did the synthetic methyl esters of keto-octadecenoic and keto-octadecadienoic acids. Dicumarol exerted a protective action against NE. The possibility is discussed that conjugated keto-polyenoic fatty acids, provided by oxidized oils or formed endogenously in vitamin E deficiency, may play a role in causing NE. © 1979 American Oil Chemists' Society.
Scientific Publication
You may also be interested in