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פותח על ידי קלירמאש פתרונות בע"מ -
Mechanisms Modulating Postharvest Pathogen Colonization of Decaying Fruits
Year:
2010
Source of publication :
Postharvest pathology
Authors :
אלקן, נעם
;
.
ברד, שירי
;
.
דוידזון, מעין
;
.
הורוביץ, סיגל בראון
;
.
ליכטר, אמנון
;
.
מיארה, איתי
;
.
פרוסקי, דב
;
.
קובילר, אילנה
;
.
שרמן, עמיר
;
.
Volume :
Co-Authors:

Robert Fluhr,Department of Plant GeneticsThe Weizmann Institute of ScienceRehovotIsrael

 

Facilitators :
From page:
43
To page:
55
(
Total pages:
13
)
Abstract:

As biotrophs, insidious fungal infections of postharvest pathogens remain quiescent during fruit growth while at a particular phase during fruit ripening and senescence the pathogens transform to necrotrophs causing typical decay symptoms. Exposure of unripe hosts to pathogens (hemi-biotroph or necrotrophs), initiates defensive signal-transduction cascades that limit fungal growth and development. Exposure to the same pathogens during ripening and storage activates a substantially different signalling cascade which facilitates fungal colonization. This chapter will focus on modulation of postharvest host-pathogen interactions by pH and the consequences of these changes. Host pH can be raised or lowered in response to host signals, including alkalization by ammonification of the host tissue as observed in Colletotrichum and Alternaria, or acidification by secretion of organic acids as observed in PenicilliumBotrytis and Sclerotinia. These changes sensitize the host and activate transcription and secretion of fungal hydrolases that promote maceration of the host tissue. Several particular examples of coordinated responses which follow this scheme are described.

Note:
Access to full text is only for authorized persons
biotrophic stage
Host acidification
Host alkalinization
Latent infections
necrotrophic colonization
pathogenicity effectors
Quiescent infections
עוד תגיות
תוכן קשור
More details
DOI :
Article number:
0
Affiliations:
Database:
גוגל סקולר
Publication Type:
פרק מתוך ספר
;
.
Language:
אנגלית
Editors' remarks:
ID:
37257
Last updated date:
02/03/2022 17:27
Creation date:
13/09/2018 14:51
You may also be interested in
Scientific Publication
Mechanisms Modulating Postharvest Pathogen Colonization of Decaying Fruits

Robert Fluhr,Department of Plant GeneticsThe Weizmann Institute of ScienceRehovotIsrael

 

Mechanisms Modulating Postharvest Pathogen Colonization of Decaying Fruits

As biotrophs, insidious fungal infections of postharvest pathogens remain quiescent during fruit growth while at a particular phase during fruit ripening and senescence the pathogens transform to necrotrophs causing typical decay symptoms. Exposure of unripe hosts to pathogens (hemi-biotroph or necrotrophs), initiates defensive signal-transduction cascades that limit fungal growth and development. Exposure to the same pathogens during ripening and storage activates a substantially different signalling cascade which facilitates fungal colonization. This chapter will focus on modulation of postharvest host-pathogen interactions by pH and the consequences of these changes. Host pH can be raised or lowered in response to host signals, including alkalization by ammonification of the host tissue as observed in Colletotrichum and Alternaria, or acidification by secretion of organic acids as observed in PenicilliumBotrytis and Sclerotinia. These changes sensitize the host and activate transcription and secretion of fungal hydrolases that promote maceration of the host tissue. Several particular examples of coordinated responses which follow this scheme are described.

Scientific Publication
You may also be interested in