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אסיף מאגר המחקר החקלאי
פותח על ידי קלירמאש פתרונות בע"מ -
Mechanism of mycoparasitism
Year:
1983
Authors :
אלעד, יגאל
;
.
Volume :
Co-Authors:

I Chet

Facilitators :
From page:
35
To page:
40
(
Total pages:
6
)
Abstract:

Antagonistic Trichoderma spp. showed a chemotropic response toward the hyphae of Rhizoctonia solani. Attachment to its host fungus was either by hyphal coils, hooks or appressoria. Recognition is apparently due to the presence of lectin on the hyphae of the pathogenic fungi which binds to specific sugars on Trichoderma cell walls. Addition of galactose and fucose prevented this phenomenon. When this interaction occurred Trichoderma harzianum excreted B-1, 3glucanase and chitinase which caused a partial lysis of R. solani and S. rolfsii cell walls and enabled the antagonist to penetrate the host's cells and consume their cytoplasm. Similarly, the antagonist was able to penetrate sclerotia of J3. rolfsii and to lyse their contents.

Note:

Colloques de l'INRA no. 18

 

Related Files :
fungi
mycoparasitism
plant protection
Rhizoctonia
Rhizoctonia solani
Trichoderma
עוד תגיות
תוכן קשור
More details
DOI :
Article number:
0
Affiliations:
Database:
גוגל סקולר
Publication Type:
מאמר מתוך כינוס
;
.
Language:
אנגלית
Editors' remarks:
ID:
37608
Last updated date:
02/03/2022 17:27
Creation date:
17/10/2018 12:33
Scientific Publication
Mechanism of mycoparasitism

I Chet

Antagonistic Trichoderma spp. showed a chemotropic response toward the hyphae of Rhizoctonia solani. Attachment to its host fungus was either by hyphal coils, hooks or appressoria. Recognition is apparently due to the presence of lectin on the hyphae of the pathogenic fungi which binds to specific sugars on Trichoderma cell walls. Addition of galactose and fucose prevented this phenomenon. When this interaction occurred Trichoderma harzianum excreted B-1, 3glucanase and chitinase which caused a partial lysis of R. solani and S. rolfsii cell walls and enabled the antagonist to penetrate the host's cells and consume their cytoplasm. Similarly, the antagonist was able to penetrate sclerotia of J3. rolfsii and to lyse their contents.

Colloques de l'INRA no. 18

 

Scientific Publication
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