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פותח על ידי קלירמאש פתרונות בע"מ -
Mode of action of Trichoderma harzianum T39: Competition, Control of Pathogen Pathogenicity Enzymes and Induced Resistance [abstract]
Year:
1999
Source of publication :
Phytoparasitica
Authors :
אלעד, יגאל
;
.
לוי, תמי
;
.
קירשנר, בני
;
.
קפט, ארנב
;
.
רב דוד, דליה
;
.
Volume :
27 (1)
Co-Authors:

E. Govrin, A. Levine, G. De Meyer and M. Hoefte

 

Facilitators :
From page:
67
To page:
68
(
Total pages:
2
)
Abstract:

Biological control of of gray mold (Botrytis cinerea) and of white mold (Sclerotinia sclerotiorum)
can be achieved by Trichoderma harzianum T39. This biocontrol agent is used for the control of B.
cinerea on grapes, tor the control of both diseases on greenhouse crops, as well as for the control of
other diseases. The biocontrol mechanism by which T. harzianum T39 is active was investigated.
T. harzianum T39 inhibits the conidial germination and germ tube elongation of B. cinerea by
competition for sources such as nutrients and space that are in short supply Ibr the pathogen.
Inhibitory compounds (antibiosis) or fungal cell wall-degrading enzymes (mycoparasitism) are not
involved in the biocontrol process. Although after one day it is expected that the germ-tubes would
penetrate the host tissue, penetration is also suppressed. This indicates another possible mechanism.
In the presence of Z harzianum T39, the activity of the hydrolytic enzymes cutin esterase, exo- and
endo-polygalacturonase, pectin methyl esterase and pectate lyase on B. cinerea infected bean leaves is
reduced. Carboxymethyl cellulase was not affected by T. harzianum T39. A serine protease currently
isolated and characterized, which originates from T39, was isolated and tbund to be responsible for
restraining the pathogen's pathogenicity. The fact that dead cells of T. harzianum T39 applied to the
leaves or live cells to the root zone of plants resulted in the reduction of foliar diseases, points to the
involvement of induced resistance.

Note:
Related Files :
Biocontrol agents
Botrytis
Botrytis cinerea
Grapes
Induced resistance
Trichoderma
Trichoderma harzianum T39
White mold
עוד תגיות
תוכן קשור
More details
DOI :
Article number:
0
Affiliations:
Database:
גוגל סקולר
Publication Type:
מאמר מתוך כינוס
;
.
תקציר
;
.
Language:
אנגלית
Editors' remarks:
ID:
37635
Last updated date:
02/03/2022 17:27
Creation date:
23/10/2018 11:16
You may also be interested in
Scientific Publication
Mode of action of Trichoderma harzianum T39: Competition, Control of Pathogen Pathogenicity Enzymes and Induced Resistance [abstract]
27 (1)

E. Govrin, A. Levine, G. De Meyer and M. Hoefte

 

Biological control of of gray mold (Botrytis cinerea) and of white mold (Sclerotinia sclerotiorum)
can be achieved by Trichoderma harzianum T39. This biocontrol agent is used for the control of B.
cinerea on grapes, tor the control of both diseases on greenhouse crops, as well as for the control of
other diseases. The biocontrol mechanism by which T. harzianum T39 is active was investigated.
T. harzianum T39 inhibits the conidial germination and germ tube elongation of B. cinerea by
competition for sources such as nutrients and space that are in short supply Ibr the pathogen.
Inhibitory compounds (antibiosis) or fungal cell wall-degrading enzymes (mycoparasitism) are not
involved in the biocontrol process. Although after one day it is expected that the germ-tubes would
penetrate the host tissue, penetration is also suppressed. This indicates another possible mechanism.
In the presence of Z harzianum T39, the activity of the hydrolytic enzymes cutin esterase, exo- and
endo-polygalacturonase, pectin methyl esterase and pectate lyase on B. cinerea infected bean leaves is
reduced. Carboxymethyl cellulase was not affected by T. harzianum T39. A serine protease currently
isolated and characterized, which originates from T39, was isolated and tbund to be responsible for
restraining the pathogen's pathogenicity. The fact that dead cells of T. harzianum T39 applied to the
leaves or live cells to the root zone of plants resulted in the reduction of foliar diseases, points to the
involvement of induced resistance.

Scientific Publication
You may also be interested in